The child convulses after crying

Children may have convulsions after crying loudly. This is a reaction caused by excessive emotional excitement. Parents do not need to be too nervous. They should pay more attention to observing their children's physical changes. If other symptoms occur, they should take them to the hospital for treatment. This may not be caused by too many emotional fluctuations. At this time, appropriate treatment measures should be found.

Hemifacial spasm, also known as clonic facial spasm, is characterized by twitching contractions of the facial muscles on one side[1]. The disease can occur at any age, including children, but it is more common in middle-aged women. Idiopathic cases are more common, or they may be temporary or permanent sequelae of idiopathic facial nerve paralysis.

The cause of hemifacial twitching is unclear, but it may be caused by ectopic excitation or pseudo-synaptic transmission of the facial nerve. Surgical and autopsy materials have confirmed that facial muscle spasm is caused by some kind of compression that causes pathological interference in the conduction of the facial nerve. In most patients, it is caused by normal vascular cross-compression, such as compression of the posterior inferior cerebellar artery, the anterior inferior cerebellar artery, and the vertebral artery.

Occasionally, it is caused by compression of the facial nerve root by an aneurysm, arteriovenous malformation, or brain tumor. A very small number of patients experience hemifacial muscle spasms after trauma, tumors or surgery. Pathogenesis: Mechanical compression caused by vascular crossing can squeeze nerve fibers together, causing them to lose their myelin sheath and lead to a short circuit of the action current between nerve axons. This is the pathological mechanism that causes hemifacial muscle spasms.

EEG, SPECT scan, and PET scan are available. Intracranial space-occupying lesions can be located and qualitatively diagnosed by cranial X-rays, brain CT and MRI examinations; cerebrovascular lesions can be diagnosed by cerebrovascular function monitoring, transcranial Doppler and angiography (pneumoencephalography, ventriculocerebrovascular angiography). 3D-TOF magnetic resonance angiography can clearly show the abnormal dilation or hyperplasia of blood vessels around the facial nerve, allowing us to understand the compression of the facial nerve. Therefore, this method is currently the best imaging examination method for diagnosing the cause of hemifacial twitching. Somatosensory evoked potentials and brainstem evoked potentials (auditory and visual evoked potentials) are of great significance for the localization and diagnosis of cranial nerve and muscle lesions.