Myocardial damage in children is a relatively common disease at present. Most of them can be diagnosed with cardiomyopathy, congenital heart, valvular heart disease and myocarditis. It is usually caused by infection, arrhythmia or coronary artery disease. After the onset of the disease, long sighs will often occur, the face is relatively pale, and the symptoms will be different from those of normal people. They will often feel tired, but the symptoms will not be too obvious. Myocardial damage in children is currently the most common diagnosis in cardiovascular clinical practice. It includes myocardial involvement caused by various reasons, and cannot be clearly diagnosed clinically as myocarditis, cardiomyopathy, congenital heart disease, valvular heart disease and other diseases. They are collectively referred to as myocardial damage clinically. It is often caused or complicated by infection, arrhythmia, coronary artery disease, hypoxia, poisoning (drugs and poisons), metabolic diseases, neuromuscular diseases, blood diseases, etc. 1. Symptoms The main symptoms are long sighs, shortness of breath, and slightly pale complexion; some older children may complain of precordial discomfort, chest tightness, and fatigue after activities; or there may be no symptoms. 2. Physical signs There are no obvious positive signs. Cardiac examination usually shows no obvious abnormalities, the heart borders are normal, the heart sounds are strong, and some patients may have tachycardia, bradycardia, or premature beats. 3. Laboratory Examination Serum myocardial enzymes (aspartate aminotransferase GOT, creatine phosphokinase CPK, creatine phosphokinase isoenzyme CK-MB, lactate dehydrogenase LDH) can all increase in the acute phase, but the increase of CPK and CK-MB is more meaningful for the diagnosis of myocardial injury. Serum troponin (Tn) is often negative. Electrocardiogram: Myocardial damage electrocardiogram changes are non-specific, and various types of electrocardiogram abnormalities may occur, but none of them are as severe as those of myocarditis. Electrocardiogram changes: Common mild T wave changes, ST There may be segment deviation (limb leads <0.05mV< span="">, chest leads <0.1 mV), low QRS voltage, and arrhythmias: such as occasional premature beats, type I0 or II0I atrioventricular block, intraventricular block and other milder ECG changes. (The ECG diagnostic criteria for myocarditis are: ST-T changes in 2 or more main leads (I, II, aVF, V5) dominated by R waves lasting for more than 4 days with dynamic changes, sinoatrial block, atrioventricular block, complete right or left bundle branch block, coupled rhythm, polymorphic, multi-source, paired or parallel premature beats, ectopic tachycardia caused by non-atrioventricular node and atrioventricular reentry, low voltage (except newborns) and abnormal Q waves.) Echocardiography shows normal heart structure and contractile function. Minimal pericardial effusion |
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