Neonatal renal insufficiency

After a newborn is born, the doctor will conduct a detailed examination of the baby's physical development. Generally, the baby develops well, but some babies may show symptoms of renal insufficiency. If this happens, timely measures must be taken and time must not be wasted to miss the best treatment. There are many reasons for neonatal renal insufficiency. It may be due to the prolonged delivery time, resulting in hypoxia of the baby. No matter what the cause is, it must be checked clearly before symptomatic treatment can be given.

①Neonatal acute renal failure refers to the damage to the kidneys in a short period of time due to various pathological conditions such as hypovolemia, shock, hypoxia, hypothermia, drug poisoning, etc. If a child suffers from neonatal acute renal failure, this disease requires hospitalization, with the focus on eliminating the cause and symptomatic treatment. Such as correcting hypoxemia, shock, hypothermia and preventing and treating infection. Therefore, it is recommended to go to the hospital for treatment as soon as possible.

② Neonatal renal insufficiency may be caused by congenital reasons. A color ultrasound should be done to see if there are any deformities in the kidneys, and then check the ureters, bladder, etc., and take measures to treat it after the cause is determined.

③ Factors: mainly acute tubular necrosis, the causes of which include severe dehydration, blood loss and long-term shock, ischemic acute tubular necrosis caused by misuse of vasoconstrictors; toxic acute tubular necrosis caused by drugs such as aminoglycosides (gentamicin, etc.), amphotericin, mannitol, contrast agents, low molecular weight dextran, and biological toxins (snake venom, mushroom poisoning, fish gall poisoning) and heavy metals, acute tubular necrosis caused by acute hemolytic diseases such as blood type incompatibility, mechanical injury, severe acute nephritis, blackwater fever, which often produce a large amount of hemoglobin, or a large amount of myoglobin produced by crush syndrome, or multiple myeloma, oliguric nephrotic syndrome, sulfonamides and uric acid kidney stones, etc., which cause obstructive acute tubular necrosis. Primary glomerular diseases such as rapidly progressive nephritis, severe acute nephritis, secondary nephritis such as lupus nephritis, Henoch-Schonlein purpura nephritis, Goodpasture's nephritis syndrome, acute interstitial nephritis, hemolytic uremic syndrome, renal vascular diseases such as renal artery infarction, renal vein thrombosis, malignant glomerulosclerosis, etc. can all cause acute renal failure.